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Clomiphene is a non-steroidal fertility medicine. It causes the pituitary gland to release hormones needed to stimulate ovulation (the release of an egg from the ovary). Clomiphene is used to cause ovulation in women with certain medical conditions (such as polycystic ovary syndrome) that prevent naturally occurring ovulation. Clomiphene may also be used for purposes not listed in this medication guide.


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Dose of clomiphene citrate in female infertility due to sperm inhibition. Br J Obstet Gynaecol 100: 869–903, 1997. 8. Tulloch, J.M.; Wylie, P.V.; Beale, G.J.; et al. The effect of antiepileptic drug doxylamine/amphetamine on sperm motility in humans. J Clin Endocrinol Metab 84: 1583–1591, 1991. 9. Tulloch, J.M. et al. Clomiphene buy clomiphene in canada citrate administration in the rat: preliminary reports. Acta Pharmacol Toxicol 42: 227–231, 1992. 10. Sperber, N.G.; Schmitt, R.A.; and Tulloch, J.M. Clomiphene citrate as an antiepileptic and antiandrogen for female patients with male infertility. Acta Pharmacol Toxicol 42: 621–632, 1992. 11. Chassolom, A.; E.; and Tulloch, J.M. Clinical efficacy pharmacokinetic and molecular pharmacodynamics of androclomiphene citrate (25 mg) in male and female mice. Acta Pharmacol Toxicol 42: 921–925, 1992. 12. Sperber, N.G., and Tulloch, J.M. Clomiphene citrate-induced ovulatory failure: preclinical results. Am J Obstet Gynecol 172: 937–940, 1992. 13. Haines, P.S. et al. Pharmacokinetics of levonorgestrel in rabbits. Contraception 40: 543–546, 1967. 14. Chassolom, A. et Amoxicillin capsules vegetarian al. Clinical trials on the pharmacokinetic, pharmacodynamic, and pharmacokinetic properties of clomiphene citrate in humans vitro: role of osmolality and plasma levels. J Clin Endocrinol Metab 74: 511–514, 1986a. 15. Dantzer, H. et al. Buy viagra and cialis online Clomiphene citrate: a novel, oral, oral-mucosal contraceptive for women who are unable to use hormonal contraceptives. Contraception 31: 541–550, 1986b. 16. Sperber, N.G.; Schmitt, R.A.; and Tulloch, J.M. Clomiphene citrate-induced ovulatory failure: preclinical results. Am J Obstet Gynecol 172: 937–940, 1992. 17. Dantzer, H. et al. The use of clomiphene citrate to treat hypoactive sexual desire disorder. J Clin Endocrinol Metab 73: 1553–1558, 1982. 18. Sperber, N.G., and Tulloch, J.M. The effects of clomiphene citrate on female sexual behavior and fertility. Contraception 39: 465–472, 1974. 19. Arakawa, Y. et al. A dose-response study in women receiving clomiphene citrate at a starting dose of 25 mg/day for 14 weeks. Fertil Steril 53: 1347–1351, 1992. 20. Dantzer, H.; Schmitt, R.A.; and Tulloch, J.M. Effect of clomiphene citrate on sexual behavior and fertility in men. Endocrinology 142: 705–710, 1986b. 21. Dantzer, H.; Schmitt, R.A.; and Tulloch, J.M. Comparison of preclinical and clinical data on the effect of clomiphene citrate on sexual behavior and fertility. Hum Reprod 11: 1007–1010, 1986c. 22. Sperber, N.G.; Tulloch, J.M. Clomiphene citrate pharmacokinetics in premenopausal women. Contraception 34: 755–760, 1986b. 23. Kontos, L.; Chassolom, A.; Thayil, L. et al. The effect of citalopram, an orally active antidepressant, on female Sexual Dysfunctions in the Pregnancy Outcome Study: a randomized double-blind trial. Fertil Steril 57: 515–526, 1988. 24. Dantzer, H.; Schmitt, R.A.; and Tulloch, J.M. Effects of clomiphene citrate on serum and preovulatory follicle-stimulating hormone estrone sulfate levels, oocyte viability and Buy orlistat cheap online endometrial thickness, maturation, pregnancy outcome.



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Clomiphene infertility female (C) by FSH and pulsatility, in vitro pulsatility (D). FSH values are shown in parentheses. were calculated from the slopes as described Achat de clomid en ligne by our group (7). Effect of estrogen on the gonadotropin-releasing hormone system and testicular function Sperm DNA fragmentation is the main reason for an increase in the number of spermatozoa testis. We used the FSH-releasing hormone (FSHR) test to assess sperm DNA fragmentation after treatment with exogenous estrogen or without progestin. The FSHR test was performed in the follicular fluid of 10 healthy male volunteers using a commercial FSHR kit (Nagelkerke A/S, Germany). The test was performed according to the manufacturer's instructions. assay has been described previously (10–12). The mean percentage of positive spermatozoa was assessed by serial dilution assay as described previously (10, 11). Effects of exogenous estrogen on gonadotropin-releasing hormone gene expression Gonadotropin-releasing hormone expression was examined using the RT-QPCR assay. Human embryonic was performed on sperm and cell lysates as described previously (13). Results are expressed as relative fold changes compared with control, as values obtained from the same samples of control experiments. Effects of exogenous estrogen on pituitary gonadotropin secretion The pituitary-gonadotropin-releasing hormone (PGR-R) assay was performed as described previously (14, 15). Gonadotropin-releasing hormone concentrations were measured by chemiluminescence method according to the manufacturer's instructions. Values are expressed as relative fold changes compared with normal men (16). Statistical methods Data were analyzed using SPSS software (version 19.0) and SPSS-DAT 16.0; SPSS, Chicago, IL). Differences in the results of two groups did not meet the criteria for a significant result. P value of <0.05 was considered significant. Results Effects of treatment with exogenous estrogen on sperm motility, DNA fragmentation, and spermatogenesis We studied 20 healthy, fertile men in the follicular fluid after 2-week treatment with either testosterone acetate (10 mg/d) or placebo (P < 0.05). There was a significant increase in the number of sperm motile (P clomiphene citrate for female infertility < 0.001) and spermatozoa in the follicular fluid after a 2-wk treatment with exogenous estrogen (P < 0.01) compared with control men (Figure 1A). There was no significant effect of exogenous estrogen on spermatogenesis (Figure 1B). Figure 1. View largeDownload slide Effects of treatment with exogenous estrogen on sperm number, motility, and spermatogenesis. (A) FSH-releasing hormone (FSHR) test in the follicular fluid of 20 healthy men indicated by positive, negative, and no change. The mean percentage of positive spermatozoa was assessed by serial dilution assay as described in the manufacturer's instructions. (B) Sperm DNA fragmentation in the follicular fluid of 20 healthy men after a 2-wk treatment with either testosterone acetate or placebo. The mean percentage of positive spermatozoa was assessed by serial dilution assay as described in the manufacturer's instructions. numbers of positive spermatozoa are shown in parentheses. Values were calculated from the slopes as described by our group. Testosterone acetate and placebo did not significantly affect sperm motility (Table 1). There was no effect of testosterone acetate treatment on sperm DNA fragmentation (Table 1), but testosterone acetate significantly reduced the number of spermatozoa that had a diameter of <1.7 μm in both the follicular fluid and semen samples (P < 0.005) (Figure 1C). This effect was absent in the serum samples of participants. Effect of treatment with ex.

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